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In the mouse cerebellar cortex, activation of purinergic P2Y1 receptors and AMPA receptors in Bergmann glial cells leads to glutamate-vesicular release that activates NMDA receptors in the interneurons, enhancing the inhibitory synaptic input to Purkinje cells (Rudolph et al., 2016)

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the inflammatory profile data were complemented by using targeted Q-PCR to assess the expression of members of the suppressor of cytokine signaling (SOCS) and signal transducer and activator of transcription (STAT) families (STAT1, STAT6 and SOCS3) ( Fig