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The mechanisms by which riboflavin protects the body against oxidative stress may be attributed to the glutathione redox cycle and also to other possible mechanisms such as the conversion of reduced riboflavin to the oxidised form

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However, L-arginine failed to prevent blood pressure increases and left ventricle remodeling due to chronic treatment with methyl ester of N-nitro-L-arginine, which is an inhibitor of eNOS

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