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Description

Heterogeneity within GBM (regional genetic variance and cellular hierarchies often regulated by different CSC niches) is accepted as the basis for resistance to multiple treatments (155, 156)

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Secondly, ectopic lesions facilitate chronic inflammation, creating an unfavorable environment that is associated with the release of various inflammatory factors

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Savitski, M

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(2023a) identified a natural NQO1 substrate, 2-methoxy-6-acetyl-7-methyljuglone (MAM), which has potent anti-cancer effects

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