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These observations provide a novel theoretical foundation for the treatment of OP via targeting the GM5-HT axis, indicating that suppression of gut-derived 5-HT biosynthesis may serve as a promising anabolic approach for bone health

A compensatory response resulting from an increased level of acetyl-carnitinewhich reflects cellular acetyl-CoA, a potent allosteric activator of PCwas reported in both INS-1 cells and isolated islets with moderately lowered PC [36]

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Wong RK, Palsson OS, Turner MJ, Levy RL, Feld AD, von Korff M, et al