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Notably, supplementing NADPH and using a pharmacological inhibitor of aldose reductase an enzyme using NADPH oxidase for the formation of sorbitol in high-glucose conditions - successfully restored the sensitivity to LPS-induced NET formation in high glucose conditions, while reducing high glucose-induced cytosolic ROS production, neutrophil elastase secretion and spontaneous NET release (Joshi et al., 2020) (Table 4)

The production of these adhesion molecules helps ease the migration of neutrophils and monocytes into the brain parenchyma

ApoC-III inhibits hepatic uptake of TRL remnants uptake by displacing ApoE from the lipoprotein surface

The triglycerides of chylomicrons are hydrolyzed by LPL, and free fatty acids are released

Curr Hematol Malig Rep 11(6):449-455, 2016